Keywords: β-Amyloid, Alzheimer, S Disease, Insulin, Insulin-Like Growth Factor 1, Amyloid Beta Protein, Oligomer, Somatomedin C Receptor, Alzheimer Disease, Article, Cell Membrane, Cognitive Reserve, Down Regulation, Human, Hyperinsulinemia, Protein Depletion, Protein Expression, Protein Function, Protein Protein Interaction, Signal Transduction, Animal, Metabolism, Pathology, Review, Amyloid Beta-Peptides, Alzheimer Disease Metabolism Pathology, Amyloid Beta-Peptides Metabolism, Brain Metabolism Pathology, Insulin Metabolism, Insulin-Like Growth Factor I Metabolism, ß-Amyloid,
Affiliations: *** IBB - CNR ***
Institute of Biostructure and Bioimaging, National Research Council, Viale Andrea Doria, Catania 95125, Italy
PhD Program in Neuropharmacology, University of Catania, Viale Andrea Doria, Catania 95125, Italy
Department of Formative Processes, University of Catania, Via Biblioteca 2, Catania 95100, Italy
Department of Drug Sciences, University of Catania, Viale Andrea Doria, Catania 95125, Italy
Department of Human Physiology and Pharmacology, University of Rome la Sapienza, Piazzale Aldo Moro, Rome 00185, Italy
Instituto Neurologico Mediterraneo, Neuromed Località Camerelle, Pozzilli 86077, Italy
References: Not available.
Beta-Amyloid monomer and insulin/IGF-1 signaling in Alzheimer's disease
Bruni AC, Bernardi L, Colao R, Rubino E, Smirne N, Frangipane F, Terni B, Curcio SA, Mirabelli M, Clodomiro A, Di Lorenzo R, Maletta R, Anfossi M, Gallo M, Geracitano S, Tomaino C, Muraca MG, Leotta A, Lio SG, Pinessi L, Rainero I, Sorbi S, Nee L, Milan G, Pappata S, Postiglione A, Abbamondi N, Forloni G, St George Hyslop P, Rogaeva E, Bugiani O, Giaccone G, Foncin JF, Spillantini MG, Puccio G * Worldwide distribution of PSEN1 Met146Leu mutation: A large variability for a founder mutation(1597 views) Neurology (ISSN: 0028-3878, 1526-632x, 1526-632xelectronic), 2010 Mar 9; 74(10): 798-806. Impact Factor:8.017 ViewExport to BibTeXExport to EndNote