HMGA proteins promote ATM expression and enhance cancer cell resistance to genotoxic agents(1170 views) Palmieri D, Valentino T, D'Angelo D, De Martino I, Postiglione I, Pacelli R, Croce CM, Fedele M, Fusco A
Keywords: Atm, Dna Repair, Hmga, Actin, Atm Protein, High Mobility Group A Protein, High Mobility Group A1 Protein, High Mobility Group A2 Protein, Protein P21, Unclassified Drug, Vinculin, Animal Cell, Article, Atm Gene, Cancer Relapse, Cancer Resistance, Cell Stress, Cell Transformation, Controlled Study, Dna Damage, Embryo, Enzyme Induction, Gene Expression, Genotoxicity, Human, Human Cell, Mouse, Nonhuman, Positive Feedback, Priority Journal, Prognosis, Promoter Region, Protein Expression, Protein Family, Transcription Initiation, Cell Cycle Proteins, Cell Line, Dna-Binding Proteins, Gene Expression Regulation, Neoplastic, Hmga Proteins, Mutagens, Phosphorylation, Genetic, Protein-Serine-Threonine Kinases, Tumor Suppressor Proteins, Ataxia Telangiectasia,
Affiliations: *** IBB - CNR ***
Dipartimento di Biologia e Patologia Cellulare e Molecolare, Istituto di Endocrinologia Ed Oncologia Sperimentale Del CNR, Università Degli Studi di Napoli Federico II, Via Pansini 5, 80131 Napoli, Italy
Istituto di Biostrutture e Bioimmagini Del CNR, Naples, Italy
Department of Molecular Virology, Immunology and Medical Genetics, Comprehensive Cancer Center, Ohio State University, Columbus, OH, United States
NOGEC (Naples Oncogenomic Center), CEINGE Biotecnologie Avanzate, Via Comunale Margherita, Naples, Italy
References: Not available.
HMGA proteins promote ATM expression and enhance cancer cell resistance to genotoxic agents