The role of calcium, lipid membranes and islet amyloid polypeptide in the onset of type 2 diabetes: Innocent bystanders or partners in a crime?(992 views) Milardi D, Sciacca MF, Randazzo L, Raudino A, La Rosa C
Front Endocrinol (ISSN: 1664-2392, 1664-2392linking, 1664-2392print), 2014; 5(DEC): 216-216.
Keywords: Amyloid, Calcium, Free Fatty Acids, Genetic Predisposition, Membranes, Obesity, Type Ii Diabetes Mellitus, Calpain 10, Cholesterol, Glucokinase Regulatory Protein, Insulin Receptor, Peptides And Proteins, Somatomedin C, Transcription Factor 7, Unclassified Drug, Article, Biological Phenomena And Functions Concerning The Entire Organism, Calcium Homeostasis, Cell Death, Channel Gating, Cytotoxicity, Dyshomeostasis, Familial Hypercalciuric Hypercalcemia, Gene Mutation, Human, Insulin Resistance, Lipid Membrane, Membrane Damage, Non Insulin Dependent Diabetes Mellitus, Nonhuman, Protein Aggregation, Risk Factor, Weight Gain,
Affiliations: *** IBB - CNR ***
Istituto CNR di Biostrutture e Bioimmagini-Sezione di CataniaCatania, Italy
Dipartimento di Scienze Chimiche Viale Andrea Doria 6, Università Degli Studi di CataniaCatania, Italy
References: Not available.
The role of calcium, lipid membranes and islet amyloid polypeptide in the onset of type 2 diabetes: Innocent bystanders or partners in a crime?
Not available.
The role of calcium, lipid membranes and islet amyloid polypeptide in the onset of type 2 diabetes: Innocent bystanders or partners in a crime?
No results.
The role of calcium, lipid membranes and islet amyloid polypeptide in the onset of type 2 diabetes: Innocent bystanders or partners in a crime?
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